Australian Nurses in the USAAs others said, US hospitals are very likely to hire you if you have a valid nursing license obtained in the US, have a pulse, can breathe...
permanent not receptors. fertile.
Florida Nursing 513Trp100329 Don't MD's in Florida have to provide fingerprints as part of the licensing process now? My appl. was very thorough, getting reciprocity from another state. Had to provide...
You are incorrect on this. I've seen the old study done (comiplete with photos) of the guys in the Dominican Republic who are completely deficient in Type II 5a-R. That's the enzyme that finasteride inhibits. I believe Bryan Shelton, who used to post on alt.baldspot, has the photos and the entire text of the study up on his site somewhere. After puberty, these guys have basically normal hormone levels, just less DHT--in other words, very similar to finasteride users. They are very muscular and fit and normal in every way. They need no special hormonal intervention. If you can find the following study, it has all the details:
"Male Pseudohermaphroditism Due to Steroid 5a-Reductase Deficiency", Peterson et al, The American Journal of Medicine Volume 62, February 1977, pp. 170-191.
career change....adviceAt your age, go into medicine. I've been a nurse for almost 9 years, and have been miserable most of the time. You will be worked like a dog, treated like you're dumb...
Interestingly enough, the five photos showed four pseudos and the brother of one of the psuedos who was completely normal as to 5a-R. The only one to have temporal hairline recession in the group was the normal guy. And--note this--he was also the *least* muscular guy among them.
The big difference between those who have this complete deficiency in type II 5a-R and finasteride users, of course, is that they are that way their whole lives. Also, finasteride users don't have a total deficiency in type II 5a-R. Instead the amount is reduced about 60% since finasteride competes with testosterone for the enzyme.
potency quickly
The rise in T appears to partially result from unconverted DHT, leaving more T left over, and also partially from the hypothalamus desiring a certain level of androgenic signal. I say this because sometimes guys get an over-androgenic response as the body increases T levels to compensate for lowered androgenicity; e.g., they may get a flare-up of pimples. If the body didn't make hormonal adjustments, the side effects on things like libido wouldn't be transient.
and taking to semen.
Absolutely. There are no negative effects on me whatsoever. I did notice a slight reduction in libido during the first two months or so. Then it completely normalized as the body made adjustments to compensate for the reduced DHT level. If there were any lasting side effects, I simply wouldn't take it. I would rate the transient side effects that did occur as minimal in my case. Some people (a very small minority) do have more significant side effects, and in a few cases they don't resolve until discontinuation of the medication.
I've also monitored things over the years via blood tests to see exactly what is going on. My T levels increased slightly with finasteride (current levels are: total T 875 ng-dL, free T 190.2 pg-mL, both considered to be in the upper normal range). My estrogen levels also increased the same percentage, but still remained low (31 pg-mL). A roughly equal increase in T and estradiol is to be expected since estradiol is derived via aromatization from testosterone. My DHT levels are a nice low 26 ng-dL
All of this said, I doubt I would ever take the newest anti-DHT treatment, dutasteride (Avodart), which is a far less conservative treatment than finasteride, as it inhibits both type I and type II isoenzymes, leading to a much greater reduction in DHT.
Mike Leake